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Ameliorative effect of treadmill exercise on cognitive deficits and amyloid-b pathology through the mitophagy in a mouse model of Alzheimer's disease |
Dong-hun Choi, Ki-Chun Kwon, Hyun-seoub Eom, Eun-taek Oh, Joon-Yong Cho |
Korean J Sport Sci. 2020;31(4):612-625. Published online December 31, 2020 DOI: https://doi.org/10.24985/kjss.2020.31.4.612 |
Ameliorative effect of treadmill exercise on cognitive deficits and amyloid-b pathology through the mitophagy in a mouse model of Alzheimer's disease Green Coconut Water Attenuates Oxidative Damage, Amyloid Pathology and Cognitive Deficits in a Mouse Model of Alzheimer's disease P1‐135: Deficiency of Neuronal P38A‐Mapk Attenuates Amyloid Pathology and Cognitive Deficits in Alzheimer’S Mouse Model Correction to: ``Hippocampal mutant APP and amyloid beta-induced cognitive decline, dendritic spine loss, defective autophagy, mitophagy and mitochondrial abnormalities in a mouse model of Alzheimer's disease'' Esculentoside A alleviates cognitive deficits and amyloid pathology through peroxisome proliferator-activated receptor γ-dependent mechanism in an Alzheimer's disease model Gut amyloid‐β induces cognitive deficits and Alzheimer's disease‐related histopathology in a mouse model P3‐420: Special lipid‐based diets alleviate cognitive deficits in the APPswe/PS1dE9 transgenic mouse model of Alzheimer's disease independent of brain amyloid deposition Treadmill exercise decreases amyloid-β burden possibly via activation of SIRT-1 signaling in a mouse model of Alzheimer's disease P1‐070: Absence of α7 nicotinic acetylcholine receptors (α7nAChR) improve cognitive deficits and dendritic pathology in a mouse model of Alzheimer's disease (AD) O2‐12‐03: GENETIC REDUCTION OF MTOR SIGNALING AMELIORATES ALZHEIMER'S DISEASE‐RELATED COGNITIVE DEFICITS AND AMYLOID‐B PATHOLOGY BY RESTORING HIPPOCAMPAL GENE EXPRESSION SIGNATURE |